English |
正體中文
|
简体中文
|
Items with full text/Total items : 29490/55136 (53%)
Visitors : 1498804 Online Users : 351
RC Version 7.0 © Powered By DSPACE, MIT. Enhanced by
NTU Library IR team.
Scope
All of CMUR
College of Humanities and Technology
CMU Publications
College of Dentistry
College of Life Sciences
醫學工程學院
China Medical University
College of Chinese Medicine
College of Health Care
College of Public Health
China Medical University Beikang Hospital
China Medical University Hospital
Other Faculties and Departments
College of Pharmacy
General Education Center
College of Medicine
Tips:
please add "double quotation mark" for query phrases to get precise results
please goto advance search for comprehansive author search
Adv. Search
Home
‧
Login
‧
Upload
‧
Help
‧
About
‧
Administer
CMUR
>
Items for Author
Loading...
Category
Loading community tree, please wait....
Year
Loading year class tree, please wait....
Items for Author "(Yi-Ping Lai)"
Return to Browse by Author
Showing 4 items.
Collection
Date
Title
Authors
Bitstream
[基礎醫學研究所] 會議論文
2014-06-27
Hypoxia suppresses myocardial survival through HIF-1α-IGFBP-3-dependent signaling and enhances cardiomyocyte autophagy and apoptotis via FoxO3a-induced BNIP3 expression.
(Yi-Ping Lai)
;
郭薇雯(Wei-Wen Kuo)
;
黃志揚(Chih-Yang Huang)*
[基礎醫學研究所] 會議論文
2012-04-22
17β-estradiol and/or Estrogen Receptor β Against Hypoxia-induced Autophagy and Apoptosis through Suppressing the HIF-1 α Pathway in H9c2 Cardiomyoblast Cells
(Yi-Ping Lai)
;
郭薇雯(Wei-Wen Kuo)
;
黃志揚(Chih-Yang Huang)*
[基礎醫學研究所] 會議論文
2011-09-10
Regulatory Mechanism of 17β-estradiol and/or Estrogen Receptor β on Hypoxia-induced BNIP3 Autophagic and Apoptotic Pathways in H9c2 Cardiomyoblast Cells
(Yi-Ping Lai)
;
郭薇雯(Wei-Wen Kuo)
;
黃志揚(Chih-Yang Huang)*
[基礎醫學研究所] 會議論文
2011-09-10
Regulatory Mechanism of 17β-estradiol and/or Estrogen Receptor β on Hypoxia-induced BNIP3 Autophagic and Apoptotic Pathways in H9c2 Cardiomyoblast Cells
(Yi-Ping Lai)
;
郭薇雯(Wei-Wen Kuo)
;
黃志揚(Chih-Yang Huang)*
DSpace Software
Copyright © 2002-2004
MIT
&
Hewlett-Packard
/
Enhanced by
NTU Library IR team
Copyright ©
-
Feedback